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[Heterozygous P2Y 2 R deletion in TgCRND8 mice increased ?-amyloid (A?) plaque load and soluble A?1-42 levels in the cerebral cortex and hippocampus, decreased the expression of the microglial marker CD11b in these brain regions and caused neurological deficits within 10 weeks of age, as compared to age-matched TgCRND8 mice.]. Sentence from MEDLINE/PubMed, a database of the U.S. National Library of Medicine.
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