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[Germ-line deletion of Loxl2 promotes lethality in half of newborn mice mainly associated to congenital heart defects, while Loxl2 overexpression triggers male sterility due to epididymal dysfunction caused by epithelial disorganization, fibrosis and acute inflammation.]. Sentence from MEDLINE/PubMed, a database of the U.S. National Library of Medicine.
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